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- W2045014958 abstract "Acute cardiac allograft rejection (ACAR) has been associated with a poor prognosis. The early diagnosis of ACAR necessitates the accurate detection of myocyte damage. Nuclear damage activates p53, a transcription factor that initiates apoptosis and repair. Endomyocardial biopsies ( n =25) from 10 cardiac allograft recipients were stained for nuclear p53. The biopsies were divided into rejection groups based on the grading of ACAR: group 1, grade 0; group 2, grade Ia and Ib; group 3, grades II and III. While clinical indices did not correlate with myocyte damage, significantly more myocytes in group 3 stained for nuclear p53 (2.48±0.60/mm 2 ) compared with group 1 (0.22±0.12/mm 2 ) and group 2 (0.43±0.18/mm 2 ). Increased expression of p53 in cardiac myocytes with grade II or grade III rejection provides an objective quantification as an aid in the diagnosis of ACAR." @default.
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- W2045014958 date "2008-11-01" @default.
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- W2045014958 title "Increased Expression of p53 Protein Correlates With the Extent of Myocyte Damage in Cardiac Allograft Rejection" @default.
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- W2045014958 doi "https://doi.org/10.1111/j.1751-7133.2008.00018.x" @default.
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