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- W2045142779 endingPage "1040" @default.
- W2045142779 startingPage "1028" @default.
- W2045142779 abstract "Fibrosis is a pathological feature of most chronic inflammatory diseases. Fibrosis, or scarring, is defined by the accumulation of excess extracellular matrix components. If highly progressive, the fibrotic process eventually leads to organ malfunction and death. Fibrosis affects nearly every tissue in the body. Here we discuss how key components of the innate and adaptive immune response contribute to the pathogenesis of fibrosis. We also describe how cell-intrinsic changes in important structural cells can perpetuate the fibrotic response by regulating the differentiation, recruitment, proliferation and activation of extracellular matrix-producing myofibroblasts. Finally, we highlight some of the key mechanisms and pathways of fibrosis that are being targeted as potential therapies for a variety of important human diseases." @default.
- W2045142779 created "2016-06-24" @default.
- W2045142779 creator A5012698757 @default.
- W2045142779 creator A5085255411 @default.
- W2045142779 date "2012-07-01" @default.
- W2045142779 modified "2023-10-16" @default.
- W2045142779 title "Mechanisms of fibrosis: therapeutic translation for fibrotic disease" @default.
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