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- W2045332357 abstract "The severe hereditary progeroid disorder Cockayne syndrome is a consequence of a defective transcription-coupled repair (TCR) pathway. This special mode of DNA repair aids a RNA polymerase that is stalled by a DNA lesion in the template and ensures efficient DNA repair to permit resumption of transcription and prevent cell death. Although some key players in TCR, such as the Cockayne syndrome A (CSA) and B (CSB) proteins have been identified, the exact molecular mechanism still remains illusive. A recent report provides new unexpected insights into TCR in yeast. The identification and characterisation of a novel protein co-purifying with the yeast homologue of CSB (Rad26) imposes reassessment of our current understanding of TCR in yeast. What about humans?" @default.
- W2045332357 created "2016-06-24" @default.
- W2045332357 creator A5030515369 @default.
- W2045332357 creator A5065375123 @default.
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- W2045332357 date "2002-08-22" @default.
- W2045332357 modified "2023-10-09" @default.
- W2045332357 title "When machines get stuck?obstructed RNA polymerase II: displacement, degradation or suicide" @default.
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- W2045332357 doi "https://doi.org/10.1002/bies.10150" @default.
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