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- W2045465871 endingPage "125" @default.
- W2045465871 startingPage "112" @default.
- W2045465871 abstract "Two decades after its first detection in the retina, nitric oxide (NO) continues to puzzle visual neuroscientists. While its liberation by photoreceptors remains controversial, recent evidence supports three subtypes of amacrine cells as main sources of NO in the inner retina. NO synthesis was shown to depend on light stimulation, and mounting evidence suggests that NO is a regulator of visual adaptation at different signal processing levels. NO modulates light responses in all retinal neuron classes, and specific ion conductances are activated by NO in rods, cones, bipolar and ganglion cells. Light-dependent gap junction coupling in the inner and outer plexiform layers is also affected by NO. The vast majority of these effects were shown to be mediated by activation of the NO receptor soluble guanylate cyclase and resultant cGMP elevation. This review analyzes the current state of knowledge on physiological NO signaling in the retina." @default.
- W2045465871 created "2016-06-24" @default.
- W2045465871 creator A5001535506 @default.
- W2045465871 creator A5011855879 @default.
- W2045465871 creator A5036772676 @default.
- W2045465871 date "2012-01-01" @default.
- W2045465871 modified "2023-10-16" @default.
- W2045465871 title "Nitric oxide signaling in the retina: What have we learned in two decades?" @default.
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