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• W2045471895 abstract "Abstract Apolipoprotein ∈ (ApoE)ε4 allele, a risk factor for the development of Alzheimer's disease (AD), is associated with increased amyloid deposition. We examined cerebral cortex in 68 AD cases using antibodies to βbeta;‐amyloid (Aβbeta;) peptides of different length (Aβbeta; 1−40 and Aβbeta; l−42 ) and found that the increased plaque frequency observed with ε4 genotypes may be largely attributed to an increase in Aβbeta; 1−40 ‐positive plaques. Indeed, both the number of Aβbeta; 1−40 ‐positive plaques, as well as the ratio of Aβbeta; I−40 / Aβbeta; 1−42 ‐positive plaques, increased with ε4 dosage. In contrast, the frequency of Aβbeta; 1−42 ‐immunoreactive plaques was similar for ε3/ε3,ε3/ε4 and ε4/ε4 genotypes. ApoE may influence Aβbeta; length by facilitating Aβbeta; 1−40 deposition onto Aβbeta; 1−42 ‐seeded plaques or by modulating the activity of a putative carboxypeptidase that forms Aβbeta; 1−40 from Aβbeta; 1−42 in situ." @default.
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• W2045471895 date "1996-03-01" @default.
• W2045471895 modified "2023-10-04" @default.
• W2045471895 title "A?-peptide length aid apolipoprotein E genotype in Alzheimer's disease" @default.
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• W2045471895 doi "https://doi.org/10.1002/ana.410390320" @default.
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