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- W2045581990 abstract "Macrophage migration inhibitory factor (MIF) acts as an upstream regulator of the innate and adaptive immune response and exhibits chemokine-like activities [ 1 Calandra T. Roger T. Macrophage migration inhibitory factor: a regulator of innate immunity. Nat. Rev. Immunol. 2003; 3: 791-800 Crossref PubMed Scopus (1330) Google Scholar , 2 Bernhagen J. Krohn R. Lue H. et al. MIF is a noncognate ligand of CXC chemokine receptors in inflammatory and atherogenic cell recruitment. Nat. Med. 2007; 13: 587-596 Crossref PubMed Scopus (937) Google Scholar ]. Conclusive evidence also indicates that MIF provides cardioprotection during myocardial ischemia–reperfusion (I/R) [ [3] Rassaf T. Weber C. Bernhagen J. Cardiovasc. Res. 2014; 102: 321-328 Crossref PubMed Scopus (58) Google Scholar ]. Moreover, MIF promotes neovascularization during hypoxic stress through recruitment of endothelial progenitor cells (EPC) after myocardial I/R [ [4] Simons D. Grieb G. Hristov M. et al. Hypoxia-induced endothelial secretion of macrophage migration inhibitory factor and role in endothelial progenitor cell recruitment. J. Cell. Mol. Med. 2011; 15: 668-678 Crossref PubMed Scopus (105) Google Scholar ]. However, studies in clinical settings in particular in comparison with other angiogenic cytokines are lacking." @default.
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- W2045581990 date "2015-02-01" @default.
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- W2045581990 title "Key role of MIF in the migration of endothelial progenitor cells in patients during cardiac surgery" @default.
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- W2045581990 doi "https://doi.org/10.1016/j.ijcard.2014.11.226" @default.
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