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- W2045613499 abstract "The loss of the endothelium barrier and vascular leakage play a central role in the pathogenesis of hemorrhagic fever viruses. This can be caused either directly by the viral infection and damage of the vascular endothelium, or indirectly by a dysregulated immune response resulting in an excessive activation of the endothelium. This article briefly reviews our knowledge of the importance of the disruption of the vascular endothelial barrier in two severe disease syndromes, dengue hemorrhagic fever and hantavirus pulmonary syndrome. Both viruses cause changes in vascular permeability without damaging the endothelium. Here we focus on our understanding of the virus interaction with the endothelium, the role of the endothelium in the induced pathogenesis, and the possible mechanisms by which each virus causes vascular leakage. Understanding the dynamics between viral infection and the dysregulation of the endothelial cell barrier will help us to define potential therapeutic targets for reducing disease severity." @default.
- W2045613499 created "2016-06-24" @default.
- W2045613499 creator A5034664089 @default.
- W2045613499 creator A5072639843 @default.
- W2045613499 date "2013-08-15" @default.
- W2045613499 modified "2023-10-14" @default.
- W2045613499 title "The role of endothelial activation in dengue hemorrhagic fever and hantavirus pulmonary syndrome" @default.
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