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- W2045713089 abstract "We read with great interest the article by Johannigman et al (Surgery 2000;127:390-4). They conclude that in non-responders to inhaled nitrogen oxide (iNO), alveolar recruitment through the addition of positive end expiratory pressure improved response to iNO therapy. In our lab, we have reached a similar conclusion by using a lamb model of congenital diaphragmatic hernia (CDH). In CDH, the herniated viscera present in crucial stages of lung development results in pulmonary hypoplasia, pulmonary hypertension, and a surfactant deficiency. In CDH-lambs, ventilation with perfluorocarbon-based liquid or treatment with intratracheal exogenous surfactant has been shown to improve pulmonary function, in part, by alveolar recruitment. iNO, on the other hand, has had minimal effect in CDH despite concentrations as high as 80 ppm. However, we have found that strategies that recruit alveoli (ie, liquid ventilation or exogenous surfactant therapy), restore response to nitric oxide." @default.
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- W2045713089 date "2000-11-01" @default.
- W2045713089 modified "2023-10-14" @default.
- W2045713089 title "Positive end expiratory pressure and response to inhalednitric oxide therapy" @default.
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- W2045713089 doi "https://doi.org/10.1067/msy.2000.110854" @default.
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