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• W2045796871 startingPage "975" @default.
• W2045796871 abstract "Little is known about ion channels that regulate the graded, subthreshold properties of nerve terminals. Using the calyx of Held, we demonstrate here a large presynaptic persistent Na+ current with unusually hyperpolarized activation voltage. This feature allowed the current to determine both the resting potential and resting conductance of the nerve terminal. Calyces express presynaptic glycine receptors whose activation depolarizes the synapse. We found that activation of the persistent Na+ current was an essential component in the response to glycine. This Na+ current originated at or very close to the terminal and was sustained even after trains of large spike-like depolarizations. Because Na+ channels also underlie the presynaptic action potential, we conclude that their action both triggers and modulates exocytosis through control of presynaptic membrane voltage." @default.
• W2045796871 created "2016-06-24" @default.
• W2045796871 creator A5027038334 @default.
• W2045796871 creator A5027184005 @default.
• W2045796871 date "2008-12-01" @default.
• W2045796871 modified "2023-10-15" @default.
• W2045796871 title "Control of Presynaptic Function by a Persistent Na+ Current" @default.
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• W2045796871 doi "https://doi.org/10.1016/j.neuron.2008.10.052" @default.
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