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• W2045851101 abstract "This study focused on the role of the HIV-derived viral protein, tat, in activating central nervous system (CNS)-derived endothelial cells (EC) to produce interleukin-8 (IL-8), a stimulator and chemoattractant for neutrophils and lymphocytes. Human CNS-EC treated with tat (100 ng/ml) demonstrated a 2 to 3 fold upregulation in IL-8 mRNA and protein. Tumor necrosis factor-alpha (TNF) and tat were found to act additively in upregulating IL-8 production. In contrast, transforming growth factor beta (TGF beta), appeared to down modulate tat-induced IL-8 production. These data suggest that extracellular tat, especially in the presence of TNF, may be responsible for the local production of IL-8." @default.
• W2045851101 created "2016-06-24" @default.
• W2045851101 creator A5014101140 @default.
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• W2045851101 date "1999-02-01" @default.
• W2045851101 modified "2023-10-03" @default.
• W2045851101 title "HIV-1 tat protein induces the production of interleukin-8 by human brain-derived endothelial cells" @default.
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• W2045851101 doi "https://doi.org/10.1016/s0165-5728(98)00198-2" @default.
• W2045851101 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/10376933" @default.
• W2045851101 hasPublicationYear "1999" @default.
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