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• W2046389212 abstract "Adiponectin, an adipocyte-derived cytokine, affects a number of physiological processes, including immune function and inflammation. We investigated whether globular adiponectin (gAd) affects the expression of inflammation-related genes in murine macrophages (RAW264 cells). DNA microarray analysis indicated that granulocyte colony-stimulating factor (G-CSF) showed the largest increase in expression in gAd-stimulated RAW264 cells. The gAd-induced secretion of G-CSF increased in a time- and dose-dependent manner. U0126 (MEK1/2 inhibitor) and PD98059 (MEK1 inhibitor) reduced the gAd-induced G-CSF mRNA expression and G-CSF protein production. gAd induced the phosphorylation of MEK1/2 and ERK1/2 in RAW264 cells. In addition, the gAd-induced phosphorylation of MEK1/2 and ERK1/2 was dramatically reduced by PD98059 and U0126, respectively. Collectively, these results suggest that MEK1/2–ERK1/2 signaling is involved in the adiponectin-induced secretion of G-CSF." @default.
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• W2046389212 date "2008-09-01" @default.
• W2046389212 modified "2023-10-11" @default.
• W2046389212 title "Induction of granulocyte colony-stimulating factor by globular adiponectin via the MEK–ERK pathway" @default.
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• W2046389212 doi "https://doi.org/10.1016/j.mce.2008.05.002" @default.
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