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- W2048377181 abstract "The hallmark of glaucoma is the hourglass pattern of optic nerve atrophy combined with preferential loss of the larger ganglion cells of the retina [1,2]. Currently, it is believed that a variety of causative factors, acting individually or multifactorially, is capable of triggering pathogenetic cascades leading to these lesions. The severe congenital form of glaucoma results from obstruction of aqueous humor drainage, caused primarily by developmental anomalies at the angle of anterior chamber [1,3–7]. Congenital glaucoma has an onset in the neonatal or infantile period, manifested by symptoms of increased intraocular pressure and corneal edema leading to excessive tearing, photophobia, and an enlargement of the globe, clinically known as buphthalmos. The term primary congenital glaucoma (PCG) is reserved for cases in which the only anatomic defect observed is isolated trabeculodysgenesis. This form of glaucoma is classified as isolated congenital glaucoma according to the Shaffer and Weiss classification [8] and as an isolated trabeculodysgenesis, according to the anatomic classification of developmental glaucomas as reported by Hoskins et al [9]." @default.
- W2048377181 created "2016-06-24" @default.
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- W2048377181 date "2003-12-01" @default.
- W2048377181 modified "2023-10-16" @default.
- W2048377181 title "Genetics and biochemistry of primary congenital glaucoma" @default.
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- W2048377181 doi "https://doi.org/10.1016/s0896-1549(03)00062-2" @default.
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