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• W2048388425 startingPage "64" @default.
• W2048388425 abstract "Two cardiac potassium (K+) channels are activated by pertussis toxin (PTX)-sensitive G proteins either directly or in a “membrane-delimited” manner. They are muscarinic K+(KACH) and ATP-sensitive K+(KATP) channels. KACH channels are responsible for acetylcholine (ACh)- or adenosine-induced deceleration of the heartbeat and atrioventricular conduction, while KATP channels are responsible for the ischemia-induced shortening of the cardiac action potential and possibly for the adenosine-mediated protection from ischemic damage. Distinct molecular mechanisms underlie G-protein activation of these cardiac K+ channels; the α subunit of PTX-sensitive G proteins activates the KATP channels, while βγ subunits activate the KACh channel. The physiologic significance of this heterogeneous mechanism remains to be determined." @default.
• W2048388425 created "2016-06-24" @default.
• W2048388425 creator A5084693611 @default.
• W2048388425 date "1994-03-01" @default.
• W2048388425 modified "2023-09-23" @default.
• W2048388425 title "G-protein control of cardiac potassium channels" @default.
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• W2048388425 doi "https://doi.org/10.1016/1050-1738(94)90011-6" @default.
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