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• W2048652667 startingPage "787" @default.
• W2048652667 abstract "Summary Multiple components of the immune response are involved in the initiation, progression and persistence of atherosclerosis. Interleukin (IL)-17A is produced by a broad variety of leukocytes and plays an important role in host defense. IL-17A is also involved in the pathology of several autoimmune diseases mainly via the regulation of chemokine expression and leukocyte migration to the site of inflammation. There is an increasing body of evidence indicating an association between elevated levels of IL-17A and cardiovascular diseases. Interestingly, this IL- 17A-dependent response occurs in parallel with the Th1-dominant immune response during atherogenesis. To date, the precise role of IL-17A+ cells in atherosclerosis is controversial. Several studies have suggested a pro-atherogenic role of IL-17A via the regulation of aortic macrophage numbers, Th1-related cytokines and aortic chemokine expression. However, two studies recently described anti-inflammatory effects of IL-17A on mouse plaque burden via possible regulation of aortic VCAM-1 expression and T cell content. Furthermore, an initial study using IL-17A-deficient mice demonstrated that IL-17A affects the immune composition and inflammatory phenotype of the aortic wall; however, no effects were observed on atherosclerosis. Further studies are necessary to fully address the role of IL-17A and other IL-17 family members in atherosclerosis." @default.
• W2048652667 created "2016-06-24" @default.
• W2048652667 creator A5000483511 @default.
• W2048652667 creator A5051060828 @default.
• W2048652667 date "2011-01-01" @default.
• W2048652667 modified "2023-09-27" @default.
• W2048652667 title "Current views on the functions of interleukin-17A-producing cells in atherosclerosis" @default.
• W2048652667 cites W1592394199 @default.
• W2048652667 cites W1805155421 @default.
• W2048652667 cites W1951642926 @default.
• W2048652667 cites W1965710704 @default.
• W2048652667 cites W1968350849 @default.
• W2048652667 cites W1970516435 @default.
• W2048652667 cites W1975204978 @default.
• W2048652667 cites W1976444920 @default.
• W2048652667 cites W1979036381 @default.
• W2048652667 cites W1984804536 @default.
• W2048652667 cites W1986868330 @default.
• W2048652667 cites W1991130533 @default.
• W2048652667 cites W1993273784 @default.
• W2048652667 cites W1995330271 @default.
• W2048652667 cites W1996866382 @default.
• W2048652667 cites W2001564659 @default.
• W2048652667 cites W2003193394 @default.
• W2048652667 cites W2004186675 @default.
• W2048652667 cites W2006249161 @default.
• W2048652667 cites W2008026482 @default.
• W2048652667 cites W2008960002 @default.
• W2048652667 cites W2010506461 @default.
• W2048652667 cites W2014260710 @default.
• W2048652667 cites W2015347941 @default.
• W2048652667 cites W2015536043 @default.
• W2048652667 cites W2016827757 @default.
• W2048652667 cites W2020233428 @default.
• W2048652667 cites W2024173356 @default.
• W2048652667 cites W2032207767 @default.
• W2048652667 cites W2033088918 @default.
• W2048652667 cites W2037062908 @default.
• W2048652667 cites W2039467806 @default.
• W2048652667 cites W2049372707 @default.
• W2048652667 cites W2053388673 @default.
• W2048652667 cites W2056809047 @default.
• W2048652667 cites W2058172577 @default.
• W2048652667 cites W2060527543 @default.
• W2048652667 cites W2065136791 @default.
• W2048652667 cites W2069227990 @default.
• W2048652667 cites W2069571875 @default.
• W2048652667 cites W2071068494 @default.
• W2048652667 cites W2075168533 @default.
• W2048652667 cites W2082602434 @default.
• W2048652667 cites W2084052630 @default.
• W2048652667 cites W2086831075 @default.
• W2048652667 cites W2091653067 @default.
• W2048652667 cites W2093897672 @default.
• W2048652667 cites W2096626379 @default.
• W2048652667 cites W2098537370 @default.
• W2048652667 cites W2100963925 @default.
• W2048652667 cites W2105825555 @default.
• W2048652667 cites W2107369528 @default.
• W2048652667 cites W2109666345 @default.
• W2048652667 cites W2114180142 @default.
• W2048652667 cites W2117047064 @default.
• W2048652667 cites W2117758842 @default.
• W2048652667 cites W2117908470 @default.
• W2048652667 cites W2121395252 @default.
• W2048652667 cites W2122148738 @default.
• W2048652667 cites W2122939321 @default.
• W2048652667 cites W2123093300 @default.
• W2048652667 cites W2126009024 @default.
• W2048652667 cites W2130140108 @default.
• W2048652667 cites W2142017607 @default.
• W2048652667 cites W2142877732 @default.
• W2048652667 cites W2143330294 @default.
• W2048652667 cites W2143786724 @default.
• W2048652667 cites W2147779949 @default.
• W2048652667 cites W2148226248 @default.
• W2048652667 cites W2151566115 @default.
• W2048652667 cites W2151826047 @default.
• W2048652667 cites W2152882892 @default.
• W2048652667 cites W2154342692 @default.
• W2048652667 cites W2155779547 @default.
• W2048652667 cites W2159198711 @default.
• W2048652667 cites W2160883911 @default.
• W2048652667 cites W2162775213 @default.
• W2048652667 cites W2163851558 @default.
• W2048652667 cites W2164280326 @default.
• W2048652667 cites W2164521200 @default.
• W2048652667 cites W2167962196 @default.
• W2048652667 cites W2169817304 @default.
• W2048652667 cites W2772222670 @default.
• W2048652667 cites W4292172722 @default.
• W2048652667 cites W4313333839 @default.
• W2048652667 doi "https://doi.org/10.1160/th11-05-0342" @default.
• W2048652667 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3323667" @default.
• W2048652667 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21946932" @default.
• W2048652667 hasPublicationYear "2011" @default.
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