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- W2049583639 abstract "Abstract It is well-established that insulin-producing pancreatic beta cells are central in diabetes. In type 1 diabetes, beta cells are destroyed by an autoimmune mechanism, whereas in type 2 diabetes, there is a decrease in functional beta-cell mass. In this context, studying beta cells is of major importance. Beta cells represent only 1% of total pancreatic cells and are found dispersed in the pancreatic gland. During the past decades, many tools and approaches have been developed to study rodent beta cells that efficiently pushed the field forward. However, rodent and human beta cells are not identical, and our knowledge of human beta cells has not progressed as quickly as our understanding of rodent beta cells. We believe that one of the reasons for this inefficient progress is the difficulty of accessing unlimited sources of functional human pancreatic beta cells. The main focus of this review concerns recent strategies to generate new sources of human pancreatic beta cells." @default.
- W2049583639 created "2016-06-24" @default.
- W2049583639 creator A5007387317 @default.
- W2049583639 creator A5008916739 @default.
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- W2049583639 date "2012-12-19" @default.
- W2049583639 modified "2023-10-14" @default.
- W2049583639 title "Concise Review: In Search of Unlimited Sources of Functional Human Pancreatic Beta Cells" @default.
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- W2049583639 doi "https://doi.org/10.5966/sctm.2012-0120" @default.
- W2049583639 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3659747" @default.
- W2049583639 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23283495" @default.
- W2049583639 hasPublicationYear "2012" @default.
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