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- W2050091551 abstract "With the widespread use of therapeutic monoclonal antibodies in the treatment of patients with cancer, resistance to these agents has become a major issue. Preclinical models of drug action or resistance have contributed to unravel the main mechanisms of resistance, involving both tumor-associated and host related factors. However our understanding of how a monoclonal antibody destroys cancer cells in a patient and why it one day stops being effective are still far from being complete. This review focuses on the available data on mechanisms of action and resistance to rituximab, and includes some additional information for other monoclonal antibodies. Innovative approaches designed to overcome resistance, such as combination immunotherapy, costimulation with cytokines or growth factors are presented." @default.
- W2050091551 created "2016-06-24" @default.
- W2050091551 creator A5006200908 @default.
- W2050091551 creator A5006793451 @default.
- W2050091551 creator A5067770954 @default.
- W2050091551 date "2009-05-01" @default.
- W2050091551 modified "2023-10-15" @default.
- W2050091551 title "Understanding and circumventing resistance to anticancer monoclonal antibodies" @default.
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- W2050091551 doi "https://doi.org/10.4161/mabs.1.3.8292" @default.
- W2050091551 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2726592" @default.
- W2050091551 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/20065642" @default.
- W2050091551 hasPublicationYear "2009" @default.
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