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- W2050151609 endingPage "10" @default.
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- W2050151609 abstract "Alzheimer's disease is characterized pathologically by extracellular senile plaques, intracellular neurofibrillary tangles, and granulovacuolar degeneration. It has been debated whether these hallmark lesions are markers or mediators of disease progression, and numerous paradigms have been proposed to explain the appearance of each lesion individually. However, the unfaltering predictability of these lesions suggests a single pathological nidus central to disease onset and progression. One of the earliest pathologies observed in Alzheimer's disease is endocytic dysfunction. Here we review the recent literature of endocytic dysfunction with particular focus on disrupted lysosomal fusion and propose it as a unifying hypothesis for the three most-studied lesions of Alzheimer's disease." @default.
- W2050151609 created "2016-06-24" @default.
- W2050151609 creator A5037446931 @default.
- W2050151609 creator A5071638745 @default.
- W2050151609 date "2012-01-01" @default.
- W2050151609 modified "2023-09-23" @default.
- W2050151609 title "Lysosomal Fusion Dysfunction as a Unifying Hypothesis for Alzheimer's Disease Pathology" @default.
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