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- W2050804711 abstract "Hypoglycemia developed during respiratory alkalosis in fasted rats. This hypoglycemia was markedly potentiated by the simultaneous injection of inhibitors of hepatic gluconeogenesis or a β-adrenergic blocking agent; was not influenced by anti-insulin serum; was attenuated by hexamethonium; and was abolished by an α-adrenergic blocking agent. The rate of removal of injected glucose by peripheral tissues increased during alkalosis in insulin-deficient rats. The uptake of [14C]-glucose by th eadipose tissue in vivo, which is stimulated by a very minute amount of insulin, was also stimulated during alkalosis whether or not the circulating insulin was neutralized with anti-insulin serum. It was concluded that, in alkalotic rats, blood glucose is rapidly utilized by peripheral tissues dependent on α-adrenergic stimulation, but without mediation of insulin and that this leads to development of hypoglycemia." @default.
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- W2050804711 title "Hypoglycemia induced by α-adrenergic stimulation during alkalosis" @default.
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- W2050804711 doi "https://doi.org/10.1016/0014-2999(77)90198-4" @default.
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