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- W2050850192 abstract "The objective of the study is to explore topographic changes of cerebral glucose metabolism (CMglc) using amyloid-beta pathology using Pittsburgh Compound B ([11C]PIB)-positron emission tomography (PET) and [18F]-fluoro-2-deoxyglucose ([18F]FDG)- PET in the amnestic mild cognitive impairment (MCI). 22 cognitively normal elderly (NL) and 12 amnestic MCI received clinical, neuropsychological, magnetic resonance imaging (MRI), PIB, and FDG-PET exams. Parametric images of PIB binding uptake and CMglc were constructed and 3D-isocontour volume-of-interests (VOIs) were drawn for group comparisons. Amnestic MCI showed significantly increased PIB binding uptake in the bilateral middle and inferior temporal, right supramarginal, right superior and inferior parietal, bilateral middle occipital, right superior occipital, bilateral median cingulate and paracingulate gyrus, bilateral amygdale, right cuneus, bilateral precuneus compared to NL (p < 0.05 for all). Compared to NL, CMglc in amnestic MCI subjects showed significant decrease only in the left inferior parietal lobule (p < 0.05). These results first support the fact that amnestic MCI is the preclinical state of AD. In addition, considerable topographical differences between amyloid deposition and decreased glucose metabolism founded in this study indicate that amyloid pathology itself does not cause directly the functional decline in MCI." @default.
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- W2050850192 date "2009-07-01" @default.
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- W2050850192 title "IC-P-044: Imaging of amyloid plaques and cerebral glucose metabolism in amnestic mild cognitive impairment" @default.
- W2050850192 doi "https://doi.org/10.1016/j.jalz.2009.05.064" @default.
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