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- W2051200768 abstract "To the Editor. — Endothelin-1 (ET-1) is a 21-residue vasoconstrictor-pressor peptide originally isolated and sequenced from porcine aortic endothelial cells. 1 We have recently demonstrated the production of ET-like immunoreactivity (LI) by renal tubular cells and the presence of ET-LI in human urine. 2 We have further shown that urinary ET-LI excretion levels remarkably increased in patients with renal disease, which originates, at least in part, from the affected renal tubules. 3 To elucidate whether urinary ET-LI is a marker for tubular injury, we measured urinary excretion levels of ET-LI in patients treated with cisplatin, a chemotherapeutic agent that often causes renal tubular damage. Study. — The present group consisted of six patients (three men and three women aged 50.0 ±6.4 years, mean± SEM) with cancers of various origins: adenocarcinoma of lung (two patients), ovarian papillary cystadenocarcinoma (two), lung metastasis of mandibular carcinoma (one), and mediastinal germ cell tumor (one)." @default.
- W2051200768 created "2016-06-24" @default.
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- W2051200768 date "1991-03-20" @default.
- W2051200768 modified "2023-10-18" @default.
- W2051200768 title "Cisplatin-Induced Urinary Endothelin Excretion" @default.
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- W2051200768 doi "https://doi.org/10.1001/jama.1991.03460110057019" @default.
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