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• W2051207831 abstract "We examined the time-course activation and the cell-type specific role of MAP kinases in puromycin aminonucleoside (PAN)-induced renal disease. The maximal activation of c-Jun-NH2-terminal kinase (JNK), extracellular signal regulated kinase (ERK), and p38 MAP kinase was detected on Days 52, 38, and 38 after PAN-treatment, respectively. p-JNK was localized in mesangial and proximal tubular cells at the early renal injury. It was expressed, therefore, in the inflammatory cells of tubulointerstitial lesions. While, p-ERK was markedly increased in the glomerular regions and macrophages p-p38 was observed in glomerular endothelial cells, tubular cells, and some inflammatory cells. The results show that the activation of MAP kinases in the early renal injury by PAN-treatment involves cellular changes such as cell proliferation or apoptosis in renal native cells. The activation of MAP kinases in infiltrated inflammatory cells and fibrotic cells plays an important role in destructive events such as glomerulosclerosis and tubulointerstitial fibrosis." @default.
• W2051207831 created "2016-06-24" @default.
• W2051207831 creator A5044956866 @default.
• W2051207831 creator A5063404969 @default.
• W2051207831 date "2004-10-01" @default.
• W2051207831 modified "2023-09-29" @default.
• W2051207831 title "Cell-type-specific activation of mitogen-activated protein kinases in PAN-induced progressive renal disease in rats" @default.
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• W2051207831 doi "https://doi.org/10.1016/j.bbrc.2004.08.047" @default.
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