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• W2051229624 abstract "The endothelium-due to its strategic anatomic position between the circulating blood and vascular smooth muscle-plays an important functional role in the coronary circulation. Endothelial cells release factors interfering with coagulation, platelet function, vascular tone, and growth. Endothelium-derived nitric oxide (NO) is the endogenous nitrovasodilator that is a potent inhibitor of platelet function and vasodilator. Together with prostacyclin, NO plays an important protective role in preventing platelet adhesion, aggregation, and coronary vasospasm. Endothelial cells also are a source of contracting factors such as endothelin-1, thromboxane A(2), and endoperoxides. Cardiovascular risk factors (hyperlipidemia, hypertension, and diabetes) inhibit the formation of endothelium-derived relaxing factors and promote that of contracting factors. In coronary arteries with advanced atherosclerosis, endothelial function is severely impaired. The reduced release of endothelium-derived NO is associated with an increased platelet-vessel-wall interaction and, in turn, platelet activation and vasospasm, both known events in coronary artery disease." @default.
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• W2051229624 date "1991-07-01" @default.
• W2051229624 modified "2023-09-27" @default.
• W2051229624 title "Endothelium-derived vasoactive factors and their role in the coronary circulation" @default.
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• W2051229624 doi "https://doi.org/10.1016/1050-1738(91)90034-c" @default.
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