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- W2051373884 abstract "Angiotensin II activates three major mitogen-activated protein kinases (MAPK) in vascular smooth muscle cells. Although other angiotensin II-induced MAPKs activation require transactivation of a growth factor receptor, the detailed mechanism by which angiotensin II activates c-Jun NH2-terminal kinase (JNK) remains unclear. Here, an immunosuppressant, cyclosporin A but not FK506, selectively inhibited angiotensin II-induced JNK activation in vascular smooth muscle cells. However, cyclosporin A had no inhibitory effect on angiotensin II-induced protein synthesis. Thus, angiotensin II-induced JNK activation but not protein synthesis is mediated by a mechanism sensitive to cyclosporin A, which is independent from calcineurin in vascular smooth muscle cells." @default.
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- W2051373884 date "2002-05-01" @default.
- W2051373884 modified "2023-09-26" @default.
- W2051373884 title "Cyclosporin A inhibits angiotensin II-induced c-Jun NH2-terminal kinase activation but not protein synthesis in vascular smooth muscle cells" @default.
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- W2051373884 doi "https://doi.org/10.1016/s0014-2999(02)01587-x" @default.
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