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• W2051702988 abstract "The prevalence of the metabolic syndrome and underlying metabolic disturbances increase rapidly in developed countries. Various molecular targets are currently under investigation to unravel the molecular mechanisms that cause these disturbances. This is done in attempt to counter or prevent the negative health consequences of the metabolic disturbances. Here, we reviewed the current knowledge on the role of C/EBP- β in these metabolic disturbances. C/EBP- β deletion in mice resulted in downregulation of hepatic lipogenic genes and increased expression of β -oxidation genes in brown adipose tissue. Furthermore, C/EBP- β is important in the differentiation and maturation of adipocytes and is increased during ER stress and proinflammatory conditions. So far, studies were only conducted in animals and in cell systems. The results found that C/EBP- β is an important transcription factor within the metabolic disturbances of the metabolic system. Therefore, it is interesting to examine the potential role of C/EBP- β at molecular and physiological level in humans." @default.
• W2051702988 created "2016-06-24" @default.
• W2051702988 creator A5006945105 @default.
• W2051702988 creator A5043854176 @default.
• W2051702988 creator A5059473457 @default.
• W2051702988 creator A5059735674 @default.
• W2051702988 date "2015-01-01" @default.
• W2051702988 modified "2023-10-17" @default.
• W2051702988 title "CCAAT/Enhancer Binding Protein<b><i>β</i></b>in relation to ER Stress, Inflammation, and Metabolic Disturbances" @default.
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