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- W2051787415 abstract "1 Respiratory and cardiovascular failure are principle toxic effects of β-blocker overdose. Respiratory arrest is the primary cause of death in β-blocker intoxicated rats. 2 The effect of β-adrenoceptor agonists on respiratory and cardiovascular failure in β-blocker overdose was investigated in a model of acute d,l-propranolol (30 mg kg -1 h -1 ) intoxication in spontaneously breathing rats. 3 Neither the aselective, hydrophilic β-agonist isoprena line (10, 25, 50 μg kg -1 min -1 ), nor the β 1 -selective, lipophilic β-agonist flerobuterol (1,3,10 μ g kg -1 min -1 ) and the β 2 -selective, lipophilic β-agonist clenbuterol (10, 25, 50 μg kg -1 min -1 ) had any beneficial effect on cardiovascular and respiratory variables or survival time in d,l-propranolol intoxicated spontaneously breathing rats. 4 Isoprenaline (10 μg kg -1 min -1 ) had no favourable effect on haemodynamic and respiratory variables in artificially ventilated d,l -propranolol intoxicated rats either. 5 Addition of dopamine to isoprenaline resulted in a significant reduction of survival time, primarily caused by a decrease in mean arterial blood pressure, in artificially ventilated d,l-propranolol intoxicated rats. Addition of glucagon to isoprenaline did not affect survival time. 6 Artificial ventilation is the most important supportive measure in d,l-propranolol intoxication in the rat." @default.
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- W2051787415 date "1996-02-01" @default.
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- W2051787415 title "Reduced survival after isoprenaline/ dopamine in d,l-propranolol intoxicated rats" @default.
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- W2051787415 doi "https://doi.org/10.1177/096032719601500204" @default.
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