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- W2051817440 abstract "The relative contribution of gluconeogenesis to the catecholamine-induced hyperglycemic response in rats remains controversial. In the present study, fasted rats were treated acutely with either ethanol or 3-mercaptopicolinic acid and then challenged later with saline, epinephrine or isoproterenol. Pretreatment with either agent suppressed the hyperglycemic response to isoproterenol more than the response to epinephrine. Isoproterenol-induced changes in blood lactate were augmented by ethanol pretreatment in normal fasted rats. In alloxan-diabetic rats, the augmented hyperglycemic response to isoproterenol was partially suppressed by pretreatment with ethanol or 3-mercaptopicolinic acid. Interestingly, pretreatment of normal, fed rats with ethanol unmasked a hyperglycemic response to isoproterenol which appeared to be the result of inhibition of insulin release. These data suggest that the hyperglycemic response to catecholamines in normal, fasted and alloxan-diabetic rats is due, in part, to stimulation of gluconeogenesis and that the response to isoproterenol in fasted rats is more dependent upon gluconeogenesis than is the response to epinephrine." @default.
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- W2051817440 date "1977-12-01" @default.
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- W2051817440 title "EFfects of ethanol and 3-mercaptopicolinic acid on isoproterenol and epinephrine-induced changes in glucose homeostasis in normal and alloxan-diabetic rats" @default.
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- W2051817440 doi "https://doi.org/10.1016/0006-2952(77)90284-2" @default.
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