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- W2052006146 abstract "Several symptoms following acute suppression of thyroid function during antihyperthyroid therapy remain yet to be uniformly recognized. We present a case manifesting electrolyte disorder, psychosis, and cardiac arrest during antihyperthyroid therapy. A 46-year-old woman diagnosed with hyperthyroidism and treated with propylthiouracil and β-blocker for a month was admitted to another hospital reporting dyspnea. A few hours after admission, she suddenly underwent cardiac arrest. After immediate cardiopulmonary resuscitation, severe hypocalcemia and cardiomegaly were diagnosed. She was transferred to our emergency room due to her obscure clinical course and severe hypocalcemia. She also experienced hallucinations. Electrocardiography, echocardiography, and chest radiography showed no abnormalities. Calcium administration gradually lowered her serum calcium to within normal range on day 5 after admission. Her mental disturbance disappeared and thyroid function returned to normal. Her severe electrolyte disorder also improved after treatment. She was discharged from the hospital in good condition. Hypocalcemia following acute suppression of thyroid function has been recently recognized as “hungry bone syndrome.” Different psychiatric symptoms and cardiocirculatory disturbances are observed during antihyperthyroid treatment. As our case showes, the importance of intensive care for patients with hyperthyroidism cannot be overemphasized." @default.
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- W2052006146 date "2001-01-01" @default.
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- W2052006146 title "Hungry Bone Syndrome, Psychosis, and Cardiac Arrest During Antihyperthyroid Therapy." @default.
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- W2052006146 doi "https://doi.org/10.3893/jjaam.12.372" @default.
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