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- W2052415298 abstract "Paroxysmal supraventricular tachycardia (SVT) is commonly produced by two mechanisms: atrial reentry via an A-V nodal pathway or a rapid firing ectopic focus. Studies in the experimental animal and in human beings with sustained SVT have shown that the site for reentry appears to be the upper A-V junction. This report documents an unusual site of reentry in a patient with recurrent SVT. Progressive intraventricular conduction delay with prolongation of the H-V interval resulted in initial conduction delay and then sustained reentry. This strongly suggests that, in man, the distal A-V conducting system may participate in a reentry circuit producing and perpetuating SVT. Paroxysmal supraventricular tachycardia (SVT) is commonly produced by two mechanisms: atrial reentry via an A-V nodal pathway or a rapid firing ectopic focus. Studies in the experimental animal and in human beings with sustained SVT have shown that the site for reentry appears to be the upper A-V junction. This report documents an unusual site of reentry in a patient with recurrent SVT. Progressive intraventricular conduction delay with prolongation of the H-V interval resulted in initial conduction delay and then sustained reentry. This strongly suggests that, in man, the distal A-V conducting system may participate in a reentry circuit producing and perpetuating SVT." @default.
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- W2052415298 date "1973-01-01" @default.
- W2052415298 modified "2023-09-26" @default.
- W2052415298 title "Reentrant Tachycardia: Participation of the Distal A-V Conduction System" @default.
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- W2052415298 doi "https://doi.org/10.1378/chest.63.1.23" @default.
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