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- W2052758095 endingPage "3314" @default.
- W2052758095 startingPage "3304" @default.
- W2052758095 abstract "Esophageal adenocarcinoma is an aggressive malignancy with a poor outcome, and its incidence continues to rise at an alarming rate. Current treatment strategies combining chemotherapy, radiation, and surgery are plagued with high rates of recurrence and metastasis. Multiple molecular pathways including the epidermal growth factor receptor, vascular endothelial growth factor, v-erb-b2 erythroblastic leukemia viral oncogene homolog (ERBB2), and Aurora kinase pathways are activated in many esophageal adenocarcinomas. In many cases, these pathways have critical roles in tumor progression. Research on the mechanisms by which these pathways contribute to disease progression has resulted in numerous biologic agents and small molecules with the potential to improve outcome. The promise of targeted therapy and personalized medicine in improving the clinical outcome is now closer than it has ever been." @default.
- W2052758095 created "2016-06-24" @default.
- W2052758095 creator A5001099941 @default.
- W2052758095 creator A5019526114 @default.
- W2052758095 creator A5043993232 @default.
- W2052758095 creator A5088976086 @default.
- W2052758095 date "2010-03-19" @default.
- W2052758095 modified "2023-10-18" @default.
- W2052758095 title "Esophageal Adenocarcinoma: Treatment Modalities in the Era of Targeted Therapy" @default.
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