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- W2053120464 abstract "During the last decade it has been shown that the central nervous system can influence the immune system. In healthy individuals, catecholamines can inhibit the production of pro-inflammatory cytokines like interleukin-6 (IL-6) and tumor necrosis factor alpha (TNF-alpha) via interaction with beta 2-adrenergic receptors. In contrast, we show here that catecholamines can stimulate the production of the interleukin-6 (IL-6) in children with the chronic inflammatory disease polyarticular juvenile rheumatoid arthritis (JRA). The induction of IL-6 is mediated by triggering of alpha 1-adrenergic receptors on peripheral blood leucocytes of the patients with polyarticular JRA. Functional alpha 1-adrenergic receptors are absent on leukocytes of normal donors and on leukocytes of patients with the oligoarticular form of the disease." @default.
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- W2053120464 date "1996-12-01" @default.
- W2053120464 modified "2023-10-17" @default.
- W2053120464 title "Functional α1-adrenergic receptors on leukocytes of patients with polyarticular juvenile rheumatoid arthritis" @default.
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- W2053120464 doi "https://doi.org/10.1016/s0165-5728(96)00125-7" @default.
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