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- W2053245087 abstract "<i>Objective:</i> Our purpose was to measure Aβ<sub>1–42</sub>, T-tau and P-tau<sub>181</sub> in the cerebrospinal fluid (CSF) of patients with posterior cortical atrophy (PCA), a presenile dementia likely to represent a variant of Alzheimer’s disease (AD). <i>Methods:</i> CSF samples from 34 subjects including 9 patients with PCA, 11 age-matched patients with AD and 14 age-matched cognitively healthy controls were analyzed using commercially available ELISA kits. <i>Results:</i> The Aβ<sub>1–42</sub>, T-tau and P-tau<sub>181</sub> levels in PCA patients differed significantly (p < 0.02) from those in healthy controls but were indistinguishable from subjects with a clinical diagnosis of AD. <i>Conclusion:</i> High T-tau and P-tau<sub>181</sub> and low Aβ<sub>1–42</sub> levels in PCA – typically observed in AD – indicate that the underlying pathology of PCA is usually AD. If these findings are replicated in PCA patients with autopsy-confirmed AD neuropathology, PCA patients may be eligible for disease-modifying AD treatments." @default.
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- W2053245087 date "2010-01-01" @default.
- W2053245087 modified "2023-09-25" @default.
- W2053245087 title "CSF-Tau and CSF-Aβ<sub>1–42</sub> in Posterior Cortical Atrophy" @default.
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- W2053245087 doi "https://doi.org/10.1159/000314679" @default.
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