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• W2053245619 abstract "1 The action of noradrenaline was studied in freshly dispersed cells of the rabbit portal vein using microelectrode techniques. 2 In normal physiological salt solution, the ionophoretic application of noradrenaline evoked an α-adrenoceptor-mediated depolarization and sometimes a β-adrenoceptor-mediated hyperpolarization. Experiments were carried out in the presence of propranolol to study the membrane mechanism associated with α-adrenoceptor activation. 3 In the current clamp mode of recording, the equilibrium potential of the noradrenaline-evoked depolarization was −1.9 mV. The depolarization was brought about by an increase in membrane conductance. 4 Under voltage clamp conditions, noradrenaline produced an inward current with a reversal potential of −7 ± 3 mV (mean ± s.e. mean). 5 The relationship between the noradrenaline-induced inward current and clamp potential was non-linear. Depolarization enhanced the conductance elicited by noradrenaline. 6 Evidence is presented which suggests that an additional conductance mechanism (probably an increase in potassium conductance) is also evoked by α-adrenoceptor stimulation in dispersed cells of rabbit portal vein." @default.
• W2053245619 created "2016-06-24" @default.
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• W2053245619 creator A5066764552 @default.
• W2053245619 date "1988-06-01" @default.
• W2053245619 modified "2023-10-16" @default.
• W2053245619 title "Mechanism of action of α-adrenoceptor activation in single cells freshly dissociated from the rabbit portal vein" @default.
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• W2053245619 doi "https://doi.org/10.1111/j.1476-5381.1988.tb11550.x" @default.
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