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• W2053290255 startingPage "135" @default.
• W2053290255 abstract "Non-apoptotic forms of programmed cell death are targets for novel approaches in anticancer therapy. Indeed, cancer cells often present with mutations in the apoptotic machinery that result in resistance to most anticancer therapies and contribute to a relatively low response rate to therapies based on the use of pro-apoptotic strategies. (Macro-)autophagy can be a highly efficient mode of cell death induction by excessive self-digestion as demonstrated by our experiments that studied the effect of radiation to induce autophagy cell death in apoptosis-deficient cells. Despite current controversies on the possible role of autophagy in the process of carcinogenesis and cancer progression by promoting cell survival, autophagy can be seen as a backup cell death mechanism, when other cell death mechanisms fail. This review will focus on the pathways linking autophagy and cancer that are relevant for target identification and on pharmaceuticals that can be utilized to improve cancer therapy by targeting the autophagic pathway." @default.
• W2053290255 created "2016-06-24" @default.
• W2053290255 creator A5021372431 @default.
• W2053290255 creator A5035724058 @default.
• W2053290255 creator A5044220823 @default.
• W2053290255 creator A5075232420 @default.
• W2053290255 date "2007-08-01" @default.
• W2053290255 modified "2023-10-01" @default.
• W2053290255 title "Autophagy signaling in cancer and its potential as novel target to improve anticancer therapy" @default.
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• W2053290255 doi "https://doi.org/10.1016/j.drup.2007.05.001" @default.
• W2053290255 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17627865" @default.
• W2053290255 hasPublicationYear "2007" @default.
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