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- W2053536427 endingPage "20" @default.
- W2053536427 startingPage "12" @default.
- W2053536427 abstract ". ▶ The physiological mechanisms of the insect CT min are poorly understood. ▶ Chill-coma is likely to be driven by an inability to maintain ionic homeostasis. ▶ Low temperature may limit insect activity through effects on ion-motive ATPases, ion channels, and/or the lipid membrane. At their critical thermal minimum (CT min ) insects enter chill-coma, a reversible state where neuromuscular transmission and movement cease. The physiological mechanisms responsible for the insect CT min remain poorly understood despite the regular use of chill-coma onset and recovery as a means to assess evolved or acquired variation in low temperature tolerance. In this review, we summarize the use of chill-coma as a metric of thermal tolerance to date, and synthesise current knowledge on the nature and plasticity of lower thermal limits to present probable physiological mechanisms of cold-induced failure. Chill-coma is likely to be driven by an inability to maintain ionic homeostasis through the effects of temperature on ion-motive ATPases, ion channel gating mechanisms, and/or the lipid membrane, leading to a loss of nerve and muscle excitability." @default.
- W2053536427 created "2016-06-24" @default.
- W2053536427 creator A5015610526 @default.
- W2053536427 creator A5057157540 @default.
- W2053536427 date "2011-01-01" @default.
- W2053536427 modified "2023-10-13" @default.
- W2053536427 title "Mechanisms underlying insect chill-coma" @default.
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- W2053536427 doi "https://doi.org/10.1016/j.jinsphys.2010.10.004" @default.