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- W2053669889 abstract "The effects of exogenous prostaglandins, inflammatory mediators known to be increased in pancreatic β-cells by IL-1β, on the replication and long-term insulin secretion by β-cells were investigated. Prostaglandins E1, E2, and F2α suppressed β-cell proliferation and long-term insulin secretion, thus mimicking the effects of IL-1β. The actions of prostaglandins were not prevented by pertussis toxin pretreatment. Additionally, the cyclooxygenase inhibitor indomethacin could not prevent the effects of IL-1β. It is concluded that prostaglandins suppress β-cell growth and long-term insulin secretion without participation of pertussis-toxin sensitive GTP-binding proteins. In addition, although their synthesis is increased by IL-1β, prostaglandins seemingly do not convey the inhibitory actions of this cytokine in the β-cell." @default.
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- W2053669889 date "1996-08-01" @default.
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- W2053669889 title "Prostaglandins inhibit pancreatic β-cell replication and long-term insulin secretion by pertussis toxin-insensitive mechanisms but do not mediate the actions of interleukin-1 β" @default.
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- W2053669889 doi "https://doi.org/10.1016/0167-4889(96)00058-4" @default.
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