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• W2053860562 abstract "The study of Hara et al. 1 Hara N. Nishiyama T. Takizawa I. et al. Decline of the red cell blood count in patients receiving androgen deprivation therapy for localized prostate cancer: impact of ADT on insulin-like growth factor 1 and erythropoesis. Urology. 2010; 75: 1441-1445 Abstract Full Text Full Text PDF PubMed Scopus (19) Google Scholar is very valuable because of the high prevalence of castration-related anemia (CRA) in patients with prostate cancer. From the study data, it is likely that the stimulation by testosterone on erythropoiesis could be supported and/or amplified by insulin-like growth factor–1 (IGF-I) at a later stage. From this view, it is not surprising that the authors have found no correlation between pre– and post–androgen deprivation therapy (ADT) hematopoietic parameters and androgen levels. Even low levels of testosterone might be able to make erythron sensitive to a subsequent growth stimulation as mediated by IGF-I, which would otherwise be refractory. It is known that testosterone directly stimulates erithropoiesis; however, it is unclear whether testosterone directly increases the IGF-I receptor expression. 2 Ratajczak J. Zhang Q. Pertusini E. et al. The role of insulin (INS) and insulin-like factor I (IGF-I) in regulating human erythropoiesis Studies in vitro under serum-free conditions—comparison to other cytokines and growth factors. Leukemia. 1998; 12: 371-381 Crossref PubMed Scopus (85) Google Scholar Decline of the Red Blood Cell Count in Patients Receiving Androgen Deprivation Therapy for Localized Prostate Cancer: Impact of ADT on Insulin-like Growth Factor-1 and ErythropoiesisUrologyVol. 75Issue 6PreviewTo elucidate the mechanism of blood hemoglobin loss in patients with prostate cancer during androgen deprivation therapy (ADT), and to examine the activity of the growth hormone (GH)/insulin-like growth factor-1 (IGF-1) axis during ADT, which plays an important role in hematopoiesis. Full-Text PDF Reply by the AuthorsUrologyVol. 76Issue 4PreviewIn selected patients with poorly controlled prostate cancer, we have a positive opinion regarding epoetin administration for anemia during androgen deprivation therapy (ADT), but we do not encourage the idea that erythropoiesis-stimulating agents (ESAs) are highly effective for those with anemia and clinically well-controlled disease. In patients with castration-resistant prostate cancer (CRPC), two previous trials have shown the advantage of ESA therapy in terms of anemia-related quality of life (QOL). Full-Text PDF" @default.
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• W2053860562 title "Re: Hara et al.: Decline of the Red Cell Blood Count in Patients Receiving Androgen Deprivation Therapy for Localized Prostate Cancer: Impact of ADT on Insulin-like Growth Factor 1 and Erythropoesis. (Urology 2010;75:1441-1445)" @default.
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• W2053860562 doi "https://doi.org/10.1016/j.urology.2010.06.059" @default.
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