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• W2055251180 endingPage "356" @default.
• W2055251180 startingPage "351" @default.
• W2055251180 abstract "Necrosis was considered to be the solo mechanism for ischemia/reperfusion (I/R)-induced cell death. Recent evidence from I/R models of the heart, liver, kidney, and brain indicates that apoptosis is a major contributor to I/R-induced cell death. However, evidence of I/R-induced apoptosis in skeletal muscle is sparse and divided. The purpose for the present study was to investigate I/R-induced necrosis and apoptosis in the cells isolated from rat skeletal muscle. A rat gracilis muscle model was used. After surgical preparation, clamps were applied on the vascular pedicle to create 4 h of ischemia and released for 24 h of reperfusion (I/R, n = 10). Clamping was omitted in sham I/R rats (sham I/R, n = 10). The muscle samples were harvested after 24 h of reperfusion for the process of cell isolation. Cells were stained by Propidium Iodide (PI) or Annexin V-FITC or both. Twenty thousand cells from each muscle sample were scanned and analyzed by flow cytometry. The average percentage of live cells was 45 ± 2% in the I/R group versus 65 ± 3% in the sham I/R group (p < 0.01). The average percentage of necrotic cells was 18 ± 1% in I/R versus 12 ± 1% in sham I/R (p < 0.01). The average percentage of apoptotic cells was 40 ± 3% in I/R versus 27 ± 3% in sham I/R (p < 0.01). Our results clearly demonstrated that I/R not only causes necrosis, but also accelerates apoptosis in the cells isolated from rat skeletal muscle. © 2007 Orthopaedic Research Society. Published by Wiley Periodicals, Inc. J Orthop Res 26:351–356, 2008" @default.
• W2055251180 created "2016-06-24" @default.
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• W2055251180 date "2008-01-01" @default.
• W2055251180 modified "2023-09-30" @default.
• W2055251180 title "Ischemia/reperfusion-induced necrosis and apoptosis in the cells isolated from rat skeletal muscle" @default.
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• W2055251180 doi "https://doi.org/10.1002/jor.20493" @default.
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