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- W2056081718 startingPage "89" @default.
- W2056081718 abstract "Human T lymphotropic virus type 1 (HTLV-1) causes a range of chronic inflammatory diseases and an aggressive malignancy of T lymphocytes known as adult T-cell leukaemia/lymphoma (ATLL). A cardinal feature of HTLV-1 infection is the presence of expanded clones of HTLV-1-infected T cells, which may persist for decades. A high viral burden (proviral load) is associated with both the inflammatory and malignant diseases caused by HTLV-1, and it has been believed that the oligoclonal expansion of infected cells predisposes to these diseases. However, it is not understood what regulates the clonality of HTLV-1 in vivo, that is, the number and abundance of HTLV-1-infected T cell clones. We review recent advances in the understanding of HTLV-1 infection and disease that have come from high-throughput quantification and analysis of HTLV-1 clonality in natural infection." @default.
- W2056081718 created "2016-06-24" @default.
- W2056081718 creator A5030896071 @default.
- W2056081718 creator A5051855810 @default.
- W2056081718 creator A5070164229 @default.
- W2056081718 date "2014-06-01" @default.
- W2056081718 modified "2023-09-23" @default.
- W2056081718 title "HTLV-1 clonality in adult T-cell leukaemia and non-malignant HTLV-1 infection" @default.
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