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- W2056105478 abstract "[Ca(2+)](i) (cytosolic [Ca(2+)]) and OS (oxidative stress) were measured simultaneously in calf pulmonary artery endothelial cells using fura-2 and carboxy-2',7'-dichlorodihydrofluorescein. ATP stimulated a [Ca(2+)](i) increase that was followed a few seconds later by an increase in OS. Pre-exposure to 5 microM H(2)O(2) potentiated these responses to ATP. Elevating or removing extracellular Ca(2+) increased or reduced the [Ca(2+)](i) response to ATP and caused parallel changes in the OS response, suggesting that this response was a consequence of the [Ca(2+)](i) response. Inhibition of mitochondria with rotenone or antimycin A affected the responses but not in a manner that allowed a simple interpretation of the role of mitochondria. These data show an intimate connection between [Ca(2+)](i) and OS that can be modulated by low levels of exogenously applied OS, allowing the possibility of positive feedback." @default.
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- W2056105478 date "2003-10-01" @default.
- W2056105478 modified "2023-10-18" @default.
- W2056105478 title "Agonist-induced calcium and oxidative stress responses in endothelial cells" @default.
- W2056105478 doi "https://doi.org/10.1042/bst0310960" @default.
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