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• W2056242893 startingPage "421" @default.
• W2056242893 abstract "Antral G-cell hyperfunction is a rare cause of hypergastrinemia, hyperchlorhydria, and duodenal ulcer disease. We found evidence for a familial basis for this disorder. The probands were two young men with aggressive duodenal ulcer who had basal and postprandial hypergastrinemia, hyperpepsinogenemia I, and basal and pentagastrin-stimulated hyperchlorhydria. All characteristics returned to normal after antrectomy and vagotomy. Antral gastrin concentrations and quantitative G-cell counts were normal, indicating hyperfunction of G-cells rather than hyperplasia. Four of 10 first-degree relatives of the two patients shared with them the combination of postprandial hypergastrinemia and hyperpepsinogenemia I. The aggregation of these abnormalities in tow families, each identified by a proband with hypergastrinemic, hyperpepsinogenemic l duodenal ulcer, suggests that antral G-cell hyperfunction may have a genetic basis." @default.
• W2056242893 created "2016-06-24" @default.
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• W2056242893 date "1981-10-01" @default.
• W2056242893 modified "2023-10-16" @default.
• W2056242893 title "Family Studies of Hypergastrinemic, Hyperpepsinogenemic I Duodenal Ulcer" @default.
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• W2056242893 doi "https://doi.org/10.7326/0003-4819-95-4-421" @default.
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