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- W2056517554 abstract "Key Points The binding of administered FVIIa to endogenous EPCR enhances its ability to bypass FVIII or FIX deficiency in vivo. EPCR modulation of function of pharmacologic FVIIa administration may be exploited in protein or gene-based FVIIa therapeutics." @default.
- W2056517554 created "2016-06-24" @default.
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- W2056517554 date "2014-08-14" @default.
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- W2056517554 title "The endothelial protein C receptor enhances hemostasis of FVIIa administration in hemophilic mice in vivo" @default.
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- W2056517554 doi "https://doi.org/10.1182/blood-2014-04-567297" @default.
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