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• W2056551384 abstract "The most well-known function of the vitamin D endocrine system is maintaining calcium and phosphorus homoeostasis, but studies in the past decades have unveiled a wide range of activities for vitamin D that extends beyond the regulation of calcium and phosphorus metabolism. Those activities include regulation of renal and cardiovascular functions and modulation of immune responses through the renin-angiotensin system (RAS) and the nuclear factor (NF)-κB pathway.2,3 The kidney has a key role in vitamin D metabolism. It not only provides the enzymatic system for the synthesis of 1,25(OH)2D, but also is involved in the uptake of filtrated 25(OH)D from urine in the form of vitamin D-binding protein (DBP)-25(OH) D complex through megalin-mediated endocytosis.4 Renal 1α-hydroxylase activity starts to decline even in the early hronic kidney disease (CKD) is a worldwide public health problem affecting more than 50 million people. Both persistently reduced glomerular filtration rate (GFR) and albuminuria are strong and independent risk factors for mortality and cardiovascular disease (CVD).1 Although the concerning increase in the prevalence of CKD has been recognized for some time, recent data indicate that the number of deaths from CKD has doubled between 1990 and 2010. Thus, the identification and treatment of risk factors for the early stages of CKD will help to prevent progression of kidney disease and reduce the risk of CVD events. In recent years, vitamin D deficiency has been recognized as a prominent feature of CKD, and there is evidence to suggest that vitamin D deficiency in turn accelerates the progression of kidney disease. However, most of these studies were conducted in high-risk populations, and there are limited longitudinal studies evaluating this issue in general populations, especially in Asia. C" @default.
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• W2056551384 date "2014-01-01" @default.
• W2056551384 modified "2023-10-15" @default.
• W2056551384 title "Molecular Mechanism Underlying the Renoprotective Action of Vitamin D" @default.
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• W2056551384 doi "https://doi.org/10.1253/circj.cj-14-0058" @default.
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