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• W2056763079 abstract "The main effects of classic calcium antagonists are mediated by the inhibition of L-type calcium channels broadly distributed within the renal vascular bed. Calcium antagonists act predominantly on the afferent arterioles, and dihydropyridines can favour the increase in glomerular hypertension and progression of kidney diseases, in particular when systemic blood pressure remains uncontrolled.Calcium antagonists have been widely used in clinical practice because of their antihypertensive capacity. The prevention of renal damage is a very important aim of antihypertensive therapy. This is particularly so taking into account the high prevalence of chronic kidney disease in the general population. Non-dihydropyridines such as verapamil have been shown to possess an antiproteinuric effect that could be particularly relevant.Recent data from clinical trials have confirmed that, in hypertensive patients with preserved renal function or with chronic kidney disease, calcium antagonists are effective antihypertensive drugs to be considered alone or in combination with an angiotensin-converting enzyme inhibitor or an angiotensin receptor blocker. In those patients presenting with proteinuric kidney disease, non-dihydropyridines could reduce proteinuria to a greater degree than dihydropyridines." @default.
• W2056763079 created "2016-06-24" @default.
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• W2056763079 date "2007-09-01" @default.
• W2056763079 modified "2023-10-14" @default.
• W2056763079 title "Are differences in calcium antagonists relevant across all stages of nephropathy or only proteinuric nephropathy?" @default.
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• W2056763079 doi "https://doi.org/10.1097/mnh.0b013e328285dfc4" @default.
• W2056763079 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17693756" @default.
• W2056763079 hasPublicationYear "2007" @default.
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