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• W2056938650 abstract "Abstract On‐resonance longitudinal relaxation time in the rotating frame ( T 1ρ ) has been shown to provide unique information during the early minutes of acute stroke. In the present study, the contributions of the different relaxation mechanisms to on‐resonance T 1ρ relaxation were assessed by determining relaxation rates (R 1ρ ) in both protein phantoms and in rat brain at 2.35, 4.7, and 9.4 T. Similar to transverse relaxation rate (R 2 ), R 1ρ increased substantially with increasing magnetic field strength ( B 0 ). The B 0 dependence was more pronounced at weak spin‐lock fields. In contrast to R 1ρ , longitudinal relaxation rate (R 1 ) decreased as a function of increasing B 0 field. The present data argue that dipole–dipole interaction forms only one pathway for T 1ρ relaxation and the contributions from other physicochemical factors need to be considered. Magn Reson Med 51:4–8, 2004. © 2003 Wiley‐Liss, Inc." @default.
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• W2056938650 date "2003-12-29" @default.
• W2056938650 modified "2023-10-16" @default.
• W2056938650 title "<i>B</i>₀dependence of the on-resonance longitudinal relaxation time in the rotating frame (<i>T</i>_1ρ) in protein phantoms and rat brain in vivo" @default.
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• W2056938650 doi "https://doi.org/10.1002/mrm.10669" @default.
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