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- W2056942935 abstract "In the most exciting genetic advances in the diagnosis of essential hypertension, genes responsible for three distinct forms of low-renin hypertension have been identified. Two of these forms are dominant: glucocorticoid remediable hypertension (a new gene created by the fusion of the 11 β-hydroxylase and aldosterone synthase genes) and Liddle's syndrome (a defect in the epithelial sodium channel). One of the forms is recessive: the syndrome of apparent mineralocorticoid excess (a defect in renal 11β-hydroxy-steroid dehydrogenase). The role of more than 20 other genes in causing hypertension has been assessed with variable findings. The most convincing evidence supports a role for the angiotensinogen gene, where linkage has been documented and an association with an intermediate phenotype of hypertension (nonmodulation) has been reported." @default.
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- W2056942935 date "1997-03-01" @default.
- W2056942935 modified "2023-10-16" @default.
- W2056942935 title "Genetic approach to diagnostic and therapeutic decisions an human hypertension" @default.
- W2056942935 doi "https://doi.org/10.1097/00041552-199703000-00015" @default.
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