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• W2057155168 abstract "Systemic changes resulting in increased levels of acute phase proteins, which accompany both acute and chronic inflammatory disorders, are determined mostly by production in hepatocytes. 1 Gabay C. Kuchner I. Acute-phase proteins and other systemic responses to inflammation. N Engl J Med. 1999; 340: 449-453 Google Scholar In the case of C-reactive protein (CRP), a 1000-fold increase may occur in response to infection, trauma, and other conditions. Peaks in CRP occur within 24–48 hours and subside within 2 weeks of an acute stimulus. 1 Gabay C. Kuchner I. Acute-phase proteins and other systemic responses to inflammation. N Engl J Med. 1999; 340: 449-453 Google Scholar Moderate changes in CRP levels have been identified with exercise or emotional stress. 2 Sorichter S. Martin M. Julius P. et al. Effects of unaccustomed and accustomed exercise on the immune response in runners. Medicine & Science in Sports & Exercise. 2006; 38: 1739-1745 Crossref PubMed Scopus (32) Google Scholar , 3 Schumacher A. Peersen K. Sommervoll L. et al. Physical performance is associated with markers of vascular inflammation in patients with coronary heart disease. European Journal of Cardiovascular Prevention & Rehabilitation. 2006; 13: 356-362 PubMed Google Scholar , 4 Ranjit N. Diez-Roux A.V. Shea S. et al. Psychosocial factors and inflammation in the multi-ethnic study of atherosclerosis. Archives of Internal Medicine. 2007; 167: 174-181 Crossref PubMed Scopus (201) Google Scholar , 5 Maes M. Delange J. Ranjan R. et al. modulation by psychotropic drugs Psychiatry Res. Acute phase proteins in schizophrenia, mania, and major depression. 1997; 66: 1-11 Google Scholar Cytokines such as interleukin-6 (IL-6), which are produced by macrophages and monocytes at the site of inflammation, stimulate production of acute phase proteins such as CRP, which appears to initiate elimination of targeted cells by its interaction with humoral and cellular effectors of inflammation. 6 Gauldie J. Richards C. Harnish D. et al. Interferon β2/B-cell stimulatory factor type 2 shares identity with monocyte-derived hepatocyte-stimulation factor and regulates the major acute phase protein response in liver cells. Proc Natl Acad Sci USA. 1987; 84: 7251-7255 Crossref PubMed Scopus (1376) Google Scholar While CRP is a nonspecific barometer of systemic inflammation, its ability to activate complement may contribute directly to inflammation. 7 Lagrand W.K. Niessen H.W. Wolbink G.J. et al. C-reactive protein colocalizes with complement in human hearts during acute myocardial infarction. Circulation. 1997; 95: 97-103 Crossref PubMed Scopus (335) Google Scholar" @default.
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• W2057155168 title "Are acute phase protein levels the link between atrial arrhythmias and inflammation?" @default.
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