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• W2057183937 startingPage "482" @default.
• W2057183937 abstract "AimsHaemostasis interrupts bleeding from disrupted blood vessels by activating platelet aggregation and coagulation. A similar mechanism termed thrombosis generates obstructive thrombi inside diseased arteries. As a consequence of this similarity, current anti-thrombotic agents increase the risk of bleeding. Atherosclerotic plaques produce significant amounts of prostaglandin E2 (PGE2), which activates its receptor EP3 on platelets and aggravates atherothrombosis. We investigated whether blocking EP3 could dissociate atherothrombosis from haemostasis." @default.
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• W2057183937 date "2013-12-09" @default.
• W2057183937 modified "2023-10-16" @default.
• W2057183937 title "Blocking the EP3 receptor for PGE2 with DG-041 decreases thrombosis without impairing haemostatic competence" @default.
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• W2057183937 doi "https://doi.org/10.1093/cvr/cvt276" @default.
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