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- W2057222012 endingPage "445" @default.
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- W2057222012 abstract "Excess visceral adipose tissue is associated with anomalies of blood glucose homoeostasis, elevation of plasma triglycerides and low levels of high-density lipoprotein cholesterol that contribute to the development of type-2 diabetes and cardiovascular syndromes. Visceral adipose tissue releases a large amount of free fatty acids and hormones/cytokines in the portal vein that are delivered to the liver. The secreted products interact with hepatocytes and various immune cells in the liver. Altered liver metabolism and determinants of insulin resistance associated with visceral adipose tissue distribution are discussed, as well as, determinants of an insulin-resistant state promoted by the increased free fatty acids and cytokines delivered by visceral adipose tissue to the liver." @default.
- W2057222012 created "2016-06-24" @default.
- W2057222012 creator A5011234349 @default.
- W2057222012 creator A5037872466 @default.
- W2057222012 date "2008-11-01" @default.
- W2057222012 modified "2023-09-30" @default.
- W2057222012 title "Impact of visceral adipose tissue on liver metabolism and insulin resistance. Part II: Visceral adipose tissue production and liver metabolism" @default.
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- W2057222012 doi "https://doi.org/10.1016/j.diabet.2008.04.002" @default.
- W2057222012 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/18562233" @default.
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