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• W2057362019 abstract "Acute vascular rejection represents a formidable barrier to clinical xenotransplantation and it is known that this type of rejection can also be initiated by xenoreactive antibodies that have limited complement-activating ability. Using a sophisticated mouse model, a recent study has provided in vivo evidence for the existence of an IgG1-mediated vascular rejection, which uniquely depends on both the activation of complement and interactions with FcγRIII on natural killer (NK) cells. Acute vascular rejection represents a formidable barrier to clinical xenotransplantation and it is known that this type of rejection can also be initiated by xenoreactive antibodies that have limited complement-activating ability. Using a sophisticated mouse model, a recent study has provided in vivo evidence for the existence of an IgG1-mediated vascular rejection, which uniquely depends on both the activation of complement and interactions with FcγRIII on natural killer (NK) cells." @default.
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• W2057362019 date "2005-01-01" @default.
• W2057362019 modified "2023-10-16" @default.
• W2057362019 title "Xenograft rejection: IgG, complement and NK cells team up to activate and destroy the endothelium" @default.
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• W2057362019 doi "https://doi.org/10.1016/j.it.2004.11.011" @default.
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