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• W2057420146 abstract "Le virus respiratoire syncytial (VRS) est responsable de 50 à 90 % des bronchiolites du nourrisson. Des facteurs liés au terrain (prématurité, pathologie associée, prédisposition), au virus (VRS A) ou environnementaux (pollution, tabagisme, …) sont déterminants dans la genèse et la gravité des bronchiolites. La mortalité de la bronchiolite va de 0,005 % à 0,2 % selon le terrain. La physiopathologie de la bronchiolite à VRS n’est pas connue. Le rôle direct du virus sur l‘épithélium, de l’immaturité du nourrisson, et, plus récemment souligné, celui de la réaction inflammatoire sont des facteurs importants. Des mécanismes immunopathologiques (anticorps IgE, réponse CD4+ Th2, …) jouent certainement un rôle dans les bronchiolites de certains enfants ou dans les formes graves. L’immunité anti-VRS n’est jamais vraiment complète, et les réinfections sont fréquentes chez les jeunes enfants et les personnes âgées où l’infection à VRS représente environ 5 % des atteintes respiratoires basses. Les infections à VPI 3 se traduisent essentiellement par des bronchiolites et à VPI 1 et 2 par des laryngotrachéites. L’expression clinique des infections à VPI 4 est peu connue. Le HMPV est responsable de bronchiolites en tous points comparables à celles dues au VRS. The respiratory syncytial virus (RSV) causes 50% to 90% of all bronchiolitis cases in infants. The occurrence and severity of bronchiolitis are dependent on patient-related factors (preterm birth, comorbidities, individual susceptibility), virus-related factors (RSV A), and environmental factors (pollution, environmental tobacco smoke, and others). Mortality rates in infants with bronchiolitis range from 0.005% to 0.2% according to patient-related factors. The pathophysiology of RSV bronchiolitis is unknown but clearly involves direct effects of the virus on the airway epithelium, airway immaturity, and recently identified effects of the inflammatory response. Immunopathologic mechanisms (e.g., IgE antibodies and CD4+ Th2 cells) play a role in some infants or in severe forms. Complete immunity to the virus is never achieved, and repeat infections are common in the young and the elderly, in whom the RSV causes about 5% of all lower respiratory tract infections. VPI3 infections usually cause bronchiolitis, whereas VPI1 and VPI2 infections manifest chiefly as laryngotracheitis. Little is known about the clinical expression of VPI4 infection. Bronchiolitis due to hMPV is indistinguishable from RSV bronchiolitis." @default.
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• W2057420146 date "2004-02-01" @default.
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• W2057420146 title "Virus respiratoire syncytial et virus para-influenza humains : clinique" @default.
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• W2057420146 doi "https://doi.org/10.1016/j.emcped.2003.06.003" @default.
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